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VERSION:2.0
PRODID:-//132.216.98.100//NONSGML kigkonsult.se iCalcreator 2.20.4//
BEGIN:VEVENT
UID:20260629T184023EDT-0931INWzfX@132.216.98.100
DTSTAMP:20260629T224023Z
DESCRIPTION:PhD Oral Defense of Dayananda Siddappa Thimmanahalli\, Departme
 nt of Animal Science\nOvarian functions are mainly regulated by complex in
 teractions of gonadotropins\, metabolic hormones and growth factors\, whic
 h are mediated through signaling networks including PI3K/Akt/MTOR and MAPK
  signaling pathways. At the level of the ovary\, granulosa/luteal cells ar
 e pivotal in integrating these signals to regulate follicular/corpus luteu
 m functions. Most of functional evidence for ovarian MTOR and MAPK signali
 ng in granulosa cells has come from in vitro studies. Recently pharmacolog
 ical inhibition model (MTOR) and conditional knock out mice model (ERK1/2)
  showed that MTOR and MAPK pathways regulate ovulation. However\, complete
  mechanistic processes of regulation ovulation by these pathways have not 
 been deciphered. Also\, roles of these pathways during follicular growth a
 s well as corpus luteum development have not been investigated.\n\nEveryon
 e in the 91ºÚÁÏÍø community is welcome to attend a PhD oral defense. Please 
 join us in celebrating the accomplishments of our PhD candidates.
DTSTART:20140724T171500Z
DTEND:20140724T171500Z
LOCATION:MS2-022\, Macdonald-Stewart Building\, CA\, QC\, St Anne de Bellev
 ue\, H9X 3V9\, 21111 Lakeshore Road
SUMMARY:PhD Oral Defense: Mechanistic Target of Rapamycin and Extra Cellula
 r Signal Regulated Kinase 1/2 in Ovarian Granulosa Cells
URL:/macdonald/channels/event/phd-oral-defense-mechani
 stic-target-rapamycin-and-extra-cellular-signal-regulated-kinase-12-ovaria
 n-237501
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